Béla Fleck And Chick Corea Announce Co-Headlining Tour With Their Full Bands

first_imgBéla Fleck and Chick Corea are no strangers to one another, touring together as a jazz duo extraordinaire. However, as Jambands reports, the two will join forces for a major full band tour from August 5-21, 2017. Fleck will bring along his band The Flecktones, while Corea will perform with his Elektric Band, who has accompanied him at several shows along his 75th birthday residency in NYC.The Flecktones made a triumphant return earlier this year, playing a handful of shows last summer for the first time in many years. Corea, meanwhile, celebrated his 75th birthday earlier this year and has shown no signs of slowing down. He’s currently in the midst of an insane residency at the Blue Note Jazz Club in NYC, which features over 80 performances.This tour is guaranteed to be an incredible musical experience, delving deep into the world of experimental jazz. The two bands are also expected to collaborate for a superjam of epic proportions as the encore of each show. The specific dates and locations have not yet been announced, but this is very exciting news! We will be sure to update once more information is revealed.last_img read more

Sleep, heart disease link leads from brain to marrow

first_img The Daily Gazette Sign up for daily emails to get the latest Harvard news. Insufficient amounts in early childhood tied to cognitive, behavioral problems What’s another hour of lost sleep? For some, a hazard Both too much and too little shut-eye can be associated with health problems Study flags later risks for sleep-deprived kids Research sheds light on body clock and links to mental health and disease As daylight saving time looms, researcher sheds light on health effects of not getting enough rest center_img Related Early birds may be happier than night owls Study identifies gene regions associated with sleep duration The research, published in February in the journal Nature, was conducted with colleagues from Brigham and Women’s Hospital, Beth Israel Deaconess Medical Center, the Medical University of Vienna, and the University of Lausanne, Switzerland. It was funded by several sources, including the National Institutes of Health and the American Heart Association.Because these results were found in laboratory mice, Swirski said the next step is to search for a similar response in people. Ultimately, he and McAlpine said, the findings highlight the importance of good sleep hygiene, while the enhanced understanding of inflammatory mechanisms could provide new avenues of investigation for other conditions in which inflammation plays a role.“If these pathways are relevant in humans, and there is reason to suspect they are, then they may be very important for possible targeting of inflammation, perhaps beyond cardiovascular disease,” Swirski said. “These pathways may be relevant in cancer, infectious disease, and many other conditions where inflammatory cells play a major role.” Researchers have known for some time that poor sleep raises heart disease risk. Now, they’ve found a chemical chain reaction that helps explain that risk, leading from poor sleep to a white blood cell surge that promotes the artery-clogging plaques of cardiovascular disease.The world’s top killer, cardiovascular disease kills 17.7 million worldwide annually, according to figures from the World Health Organization. It has been linked to a number of risk factors, including smoking, a poor diet, and lack of exercise. A less widely known risk is chronically poor sleep, whether short or fragmented, like that experienced by night-shift workers, travelers in the grip of jet lag, and sufferers of sleep apnea and similar conditions.“There are studies that suggest [sleep] can be as potent a driver of the disease as more traditional risk factors, such as smoking or high cholesterol levels,” said Cameron McAlpine, a research fellow in the lab of Filip Swirski, an associate professor of radiology at Harvard Medical School and a researcher at Massachusetts General Hospital’s Center for Systems Biology.Four years ago, McAlpine, Swirski, and their colleagues began experiments designed to explore the connection between sleep and the immune and inflammatory mechanisms that play a role in atherosclerosis.Atherosclerosis, also known as hardening of the arteries, is a key feature of heart disease and has come to be understood as largely an inflammatory condition, McAlpine said. It typically advances with age as fatty plaques deposit along the walls of blood vessels, narrowing them and interfering with blood flow. The atherosclerotic plaques are made up of fats like LDL, or “bad,” cholesterol and white blood cells that flood to the scene and become entangled in fibers that hold the plaque together. The plaques not only reduce blood flow, they also can rupture and cause blood clots that clog arteries leading to the brain, causing strokes, or the heart, causing heart attacks.“The research showing a link between sleep and cardiovascular disease in humans is abundant,” Swirski said. “We wanted to know the ‘how.’ In this study we uncovered one small piece of what is surely a much larger puzzle.”Through multiple experiments, researchers found that poor sleep causes production of a protein called hypocretin to fall in the brain’s hypothalamus region, which is responsible for wakefulness, energy levels, and sleep patterns. Low hypocretin levels stimulate the bone marrow to increase production of a second protein, called colony-stimulating factor 1 (CSF-1). CSF-1, in turn, signals the bone marrow’s blood stem cells to step up production of white blood cells, boosting the immune and inflammatory response that is a feature of the condition.In their experiments, conducted on mice fed a high-fat diet and genetically preprogrammed to develop atherosclerosis, the sleep-deprived mice had more white blood cells in their bloodstreams and developed larger plaques, and those plaques contained more white blood cells than those of control mice whose sleep wasn’t disrupted. Researchers then gave supplemental hypocretin to the sleep-deprived mice and found that the prevalence of atherosclerosis declined.“The role of hypocretin was certainly very, very shocking and unexpected to us. We really didn’t know what to make of it initially,” McAlpine said. “We had no idea we would find increasing white blood cells and this production could actually be regulated by sleep.”last_img read more

Eaves reflects on winless Border Battle

first_imgComing off of a 2-2 overtime tie Friday night and a 3-1 loss Saturday to third-ranked Minnesota, Wisconsin men’s hockey head coach Mike Eaves is looking to move forward and focus on the upcoming weekend.The Border Battle in Minneapolis took the Badgers’ winless streak to five games, their last victory coming Oct. 26 over Minnesota-Duluth. Though Wisconsin (1-5-2) battled its way to a tie with one of the top teams in the WCHA in game one, two third period goals from the Gophers in the second game of the series left the Badgers from earning their second win of the season in Minneapolis. “When you lose like we did on Saturday, where we are close, hanging around, having chances, it really hurts,” Eaves said at his Monday press conference. “On Saturday, we looked at the video at the things we didn’t like, and we didn’t pass the puck very well, we didn’t connect the dots like we’ve seen our kids do.”Despite the defeat in Mariucci Arena Saturday, Eaves said he has re-evaluated his plan for practice this week and will try out new strategies in a series with Minnesota State this weekend.“We’re going to go back and work on our skill, we’re going to go over our system details, and we’re quite frankly going to skate a little bit to make sure we’re covering all our bases so that we’re not leaving anything to doubt,” Eaves said.When asked about what is keeping the veteran forwards from taking control of the game and being productive up to this point, Eaves pointed out that several key contributors are not on the ice due to injuries and other issues. “When you take out (Mark) Zengerle and Nic Kerdiles, we have different chemistry up front,” Eaves said. “With the lack of goal production by having the forwards back, some of the chemistry is missing.”A junior forward, Zengerle has 92 points off 20 goals and 72 assists in his career and entered the 2012-13 campaign as college hockey’s top returning scorer. He is eight points shy of hitting the 100-point mark for his career, an achievement that would make him the 71st Badger in program history to reach the century mark.Zengerle had points in Wisconsin’s first five games before getting injured in the second period of the sixth game Nov. 3 and is expected to miss four-to-six weeks from the time of his injury.Due to a violation of the NCAA’s amateurism rules, Kerdiles, a highly-touted freshman forward, will first suit up for the Badgers in a road game against Denver Nov. 30.With Zengerle out, Eaves is working on creating a more balanced lineup, moving some players to, in the coach’s own words, “see if that gives us a little jump” and will review the power plays to see if any adjustments should be made.Asked if he had ever imagined his team’s potential had it not suffered the injuries and other issues keeping several of his top players off the ice, Eaves said he is trying to make the best of a difficult situation.“I think [the team] wants to know, ‘OK, we weren’t as successful as we were this weekend, what’s our plan coach, give us the truth in what you saw and let’s move forward and let’s get better and figure out what we’re going to do against Minnesota State,’” he explained.“That’s the way we’re going to look at it.”Transition on benchAfter the departure of Wisconsin assistant coach Bill Butters two weeks ago, the series against Minnesota marked Eaves’ first time with Gary Shuchuk coaching the Badger defensemen.Eaves described his communication with Schuck, another assistant coach, as “decent” but said there is certainly room to improve as the assistant adjusts to his new coaching responsibilities with the team.Though the transition has been relatively smooth so far, Eaves pledged that he will find a replacement for Butters before the season is over. Wisconsin’s head coach said Butters, who left the team to pursue a calling in the ministry, played too important a role for this team to not find a successor soon.“There’s too big a hole that would exist – I think we can kind of make do, but I think we need to fill that hole in order to cover all our bases as far as coaching, teaching, recruiting, working with these young people,” Eaves said. “So we will get somebody in. Maybe we could afford two microphones.”last_img read more

In Ellen Corkrum Case: Bility Gets Separate Trial

first_imgAfter been indicted along with several co-defendants including Ellen Corkrum, for allegedly stealing millions of United States Dollars from the Liberia Airport Authority (LAA), Mr. Musa H. Bility, will now get a separate trial.He was granted a separate trial on Tuesday, January 7, after his request for a “Motion for Severance” (separate trial) was accepted by Judge Blamo Dixon of Criminal Court ‘C’ at the Temple of Justice.Bility is the president of the Liberia Football Association (LFA) and former Board Chair of LAA.Besides, Bility and Madam Corkurm, others included First International Bank (FIB), Liberia Bank for Development and Investment (LBDI), as well as the Diaspora Consulting, and Melvin Johnson and Associates, all of the United State of America (USA).They were charged with economic sabotage, theft of property, criminal conspiracy and misapplication of entrusted property by the government.But, accepting Bility’s request, on Tuesday, Judge Dixon declared, “Musa Bility can’t be tried with the co-defendants, because, his interest and defense will clash with those of the other co defendants.”“As regards his separate trial with FIBank,” Judge Dixon noted, “No way he can be tried with the bank, because the bank has restituted the US$56,750 transferred from LAA accounts to Melvin Johnson and Associates in the US.” Judge Dixon further ruled that “Bility will not be tried along with the Liberia Bank for Development and Investment on grounds that my predecessor already granted separate trial for that Bank.”“He can’t be tried with co-defendants Ellen Corkrum and Melvin Johnson, because both of them are not in the bailiwick of the court.”“Therefore, the Motion Severance (separate trial) for Bility is hereby granted and his trial will commence on February 12,” the Criminal Court Judge ordered.Bility’s lawyer in a five-count request argued that he as chairman of the Board of Directors, Bility did not have the statutory responsibility for the day to day running of the LAA.They further argued that trying him and the other defendants in a joint trial would be greatly prejudicial to his interests.The lawyers also contended that as a matter of law and fact Bility’s defense to the crimes are separate and distinct, and therefore, were contradictory to the defense of the other named defendants. Share this:Click to share on Twitter (Opens in new window)Click to share on Facebook (Opens in new window)last_img read more